Mercola: Anyone who knows a little about bird flu is likely to wonder where Redfield and other “experts” are getting their predictions from, as natural bird flu is notoriously harmless to humans.

In early April, news of a highly pathogenic bird flu ripping through chicken and turkey flocks in the U.S., triggering the slaughter of millions of these animals, was reported.

Historically, however, the bird flu has never posed a threat to mankind — that is until scientists started tinkering with it, creating a hybrid with human pandemic potential....

https://organicconsumers.org/yoshihi...y-fauci-gates/

The state of Wisconsin is home to the most notorious virologist on the planet doing the most dangerous lab work in the world.

Yoshihiro Kawaoka at the University of Wisconsin at Madison creates diseases far more deadly than COVID-19. His experiments (and lab safety breaches) have been so scary that they triggered an Obama Administration moratorium. Since the Trump Administration lifted the moratorium, Kawaoka has resumed the very same work.

As Sam Husseini reports in “Wisconsin Bill Would Stop Lab Work that Could Cause Catastrophic Pandemic,” the state legislature might try to shut it down again with Senate Bill 401 which would prohibit institutions of higher education from conducting gain-of-function research on potentially pandemic pathogens.


Who is Yoshihiro Kawaoka?

Kawaoka was hand-picked by Anthony Fauci to take pathogens that only infect animals and use genetic engineering and synthetic biology to create pathogens that make humans sick. Since 1990, Fauci has funded Kawaoka under grants with titles including “Influenza Virus Assembly.”

Highly pathogenic bird flu didn’t cause disease in humans until this potential had been studied by a lab for several years. Then, it happened in Hong Kong in 1997, in the midst of what the British call the “Hong Kong handover,” when sovereignty over Hong Kong was transferred from the U.K. to China. It was during this “politically sensitive” year that the World Health Organization’s reference laboratory at the University of Hong Kong, confirmed human cases of H5N1 that infected 18 people and killed six.

https://web.archive.org/web/20220410...1229200738.htm

Science News: Genes That Made 1918 Flu Lethal Isolated


“By mixing and matching a contemporary flu virus with the “Spanish flu” — a virus that killed between 20 and 50 million people 90 years ago in history’s most devastating outbreak of infectious disease — researchers have identified a set of three genes that helped underpin the extraordinary virulence of the 1918 virus.

Writing December 29 in the Proceedings of the National Academy of Sciences, a team led by University of Wisconsin-Madison virologists Yoshihiro Kawaoka and Tokiko Watanabe identifies genes that gave the 1918 virus the capacity to reproduce in lung tissue, a hallmark of the pathogen that claimed more lives than all the battles of World War I combined….

To find the gene or genes that enabled the virus to invade the lungs, Kawaoka and his group blended genetic elements from the 1918 flu virus with those of a currently circulating avian influenza virus and tested the variants on ferrets, an animal that mimics human flu infection.

For the most part, substituting single genes from the 1918 virus onto the template of a much more benign contemporary virus yielded agents that could only replicate in the upper respiratory tract. One exception, however, included a complex of three genes that, acting in concert with another key gene, allowed the virus to efficiently colonize lung cells and make RNA polymerase, a protein necessary for the virus to reproduce.

“The RNA polymerase is used to make new copies of the virus,” Kawaoka explains. Without the protein, the virus is unable to make new virus particles and spread infection to nearby cells.

In the late 1990s, scientists were able to recover genes from the 1918 virus by looking in the preserved lung tissue of some of the pandemic’s victims. Using the relic genes, Kawaoka’s group was able to generate viruses that carry different combinations of the 1918 virus and modern seasonal influenza virus.

When tested, most of the hybrid viruses only infected the nasal passages of ferrets and didn’t cause pneumonia. But one did infect the lungs, and it carried the RNA polymerase genes from the 1918 virus that allowed the virus to make the key step of synthesizing its proteins.

In 2004, Kawaoka and his team identified another key gene from the 1918 virus that enhanced the pathogen’s virulence in mice. That gene makes hemagglutinin, a protein found on the surface of the virus and that confers on viral particles the ability to attach to host cells.

“Here, I think we are talking about another mechanism,” Kawaoka says. The RNA polymerase is used to make copies of the virus once it has entered a host cell. The role of hemagglutinin is to help the virus gain access to cells.””