For dudes to keep their looks at 50+, need 3 things to happen.

1. Can't get fat. It's the rare fat face/fat body that looks good.
2. Avoid this reaper. This is obviously the most challenging part of ageing. And while some baldcells can still look decent, these unicorns are rare.
3. If you can avoid the reaper, got to keep your hair not grey until your 70's. Grey hair rarely helps anyone's asthetics. Salt and pepper starting at 60 is fine.


Luckily for us dudes, having a fat wallet usually offsets all of the above.

if youre a timmy at 25 i don’t think being 35 or 45 is gonna do you any better looks-wise

the men that people describe as aging like fine wine are usually already good looking from a younger age, avoided the reaper, and kept relatively fit. think paul walker, brad pitt, tyson beckford

Just glad it doesn't matter what I look like at any age because my cock will always be big, thick and juicy. Just have to show some young ladies and they'll be slobbing it

-derma roll
-minoxidil
-azelaic acid
-take vitamin b6 and 25-50mg zinc per day

last 2:https://onlinelibrary.wiley.com/doi/...1988.tb03474.x

had a longer post but the system keeps giving me an error when I post it

Yeah the real Chads live in the north of Italy srs. We are a mix of mediterranean features and nordic height, best of both worlds

Man can cheat with TRT and HGH and still look sharp in their 40s and 50s. Ofc not too high of TRT doses, because it can make you look older. With good amount of test in your blood you will perform like 20-25 yo guy in the bed.
The biggest problem is the reaper, who sometimes won't even spare some women. I have see women with noorwood 1-2 and thin hair in their 30s and looks ****ing horrible. Being man you can just shave it, but women with thin hair look like a cancer patient who just started a chemio.

This is a very interesting article[URL=https://www.sciencedirect.com/science/article/pii/S0022202X15418470#f0020]https://www.sciencedirect.com/scienc...15418470#f0020[/URL]
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[i]
The skin is the largest organ in mammals, consisting of three layers, epidermis, dermis, and hypodermis, with associated appendages, including hair follicles (HFs), sebaceous, and sweat glands. The biology of epidermal stem cells in the skin is well known (Blanpain and Fuchs, 2009; Baker and Murray, 2012). In particular, the pilo-sebaceous unit (HF and associated glands) and the HF growth cycle constitute a very well-characterized model to study the functional regulation of skin stem cells. The HF is a complex mini-organ that invaginates from epidermal sheets of the skin into the dermis (Muller-Rover et al., 2001; Baker and Murray, 2012; Gilhar et al., 2012). Hair production is the result of a cyclic activity of the HF alternating three sequential phases: anagen (growth), catagen (cessation and regression), and telogen (rest). The HF is divided into two main regions depending on size variation rates through the hair cycle: permanent and cyclic regions. The upper permanent region, composed of the infundibulum, associated glands, and the bulge, a major reservoir of HF stem cells, regulates the activity of the dermal papilla in the cyclic region (Ramos et al., 2013). Activation of the dermal papilla cells during the anagen phase promotes extensive cell proliferation and differentiation in the cyclic region, ultimately resulting in the formation and growth of the hair shaft. Skin components are constantly renewed by a stock of multipotent epidermal stem cells, which are located in the bulge region (Bg) of the HF and in the basal layer of the interfollicular epithelium (Blanpain and Fuchs, 2009; Baker and Murray, 2012).

Important and well-known molecular mechanisms, including Wnt/β-catenin, BMP/Tgfβ/Smad, PI3K/Akt, and ERK/MAPK signaling pathways (Baker and Murray, 2012; Lopez-Pajares et al., 2013; Ramos et al., 2013), have key roles in the regulation of different aspects of skin stem cell and HF function. However, skin homeostasis and the HF cycle are extremely complex and multifactorial processes. Many of the interplaying mechanisms that govern and/or modulate the functional output of these processes are not well characterized. In particular, the potential physiological role of reactive oxygen species (ROS) in the skin is largely unknown.

The generation of ROS as by-products of essential and efficient metabolic reactions, such as cellular respiration or oxidase activity, is an inevitable biochemical side effect that can be extremely harmful for cell viability. The oxidative stress induced by the undesirable intracellular accumulation of ROS is a major cause of cell and tissue toxicity, and it is associated with several human diseases, including neurodegenerative (Parkinson’s, Alzheimer’s, and Huntington’s diseases), psychiatric (schizophrenia and bipolar disorder) and cardiovascular (stroke and myocardial infarction) disorders (Pieczenik and Neustadt, 2007; Valko et al., 2007). ROS are also implicated in sickle cell disease and the fragile X and chronic fatigue syndromes (el Bekay et al., 2007; Pieczenik and Neustadt, 2007). The so-called oxidative stress or free-radical theory of aging proposes a causal link between gradual, time-dependent ROS production during the whole lifetime and aging of the organism, a controversial theory that remains the subject of intense debate (Perez et al., 2009; Speakman and Selman, 2011).

Aerobic organisms have evolved powerful mechanisms to manage excess ROS production, and they are able to efficiently detoxify superoxide anions, hydroxyl radicals, hydrogen peroxide, and organic hydroperoxides into harmless H2O and O2. These mechanisms are exemplified by the superoxide dismutase, catalase, and glutathione peroxidase enzymatic systems (Machlin and Bendich, 1987; Fernandez and Videla, 1996; Mates and Sanchez-Jimenez, 1999). Interestingly, in the course of evolution, eukaryotic organisms have also developed systems to use ROS production for their own benefit. An example is the ****ocyte, which is an essential cell player of the mammalian immune system that can produce radical oxygen and nitrogen species in a tightly controlled way to kill target pathogens (Klebanoff et al., 1983; Bylund et al., 2010).

Accumulated evidence suggests that eukaryotic cells can also actively promote the production of small amounts of ROS as part of signaling pathways that regulate cell survival and proliferation (Droge, 2002; Chiarugi and Cirri, 2003; Bartosz, 2009; Sena and Chandel, 2012). Furthermore, it has been reported that exogenous ROS can regulate stem cell function in in vitro systems (Le Belle et al., 2010). An abnormal ROS production has been also linked to the deregulation of intestinal stem cell proliferation that occurs during colorectal cancer initiation (Myant et al., 2013). These observations imply the existence of widespread ROS-dependent mechanisms for the regulation of cell function and tissue homeostasis. Here we have used the skin and the well-characterized epidermal stem cell niche located in the Bg of the mouse HF as a working model to provide a proof-of-concept for the hypothesis that a transient modulation of in situ ROS levels can regulate skin homeostasis and stem cell function in a whole organism.[/i][i]

Here we have shown that switching on in situ ROS production can regulate functional responses in a tissue through the stimulation of cell proliferation and of a stem cell niche. Our results are in agreement with recent reports showing a role for ROS during the stimulation of neural stem cells in vitro in a neurosphere model (Le Belle et al., 2010) and in the deregulation of intestinal stem cell proliferation that occurs during colorectal cancer initiation (Myant et al., 2013). In these studies, an exogenous and/or continuous ROS supply or a systemic ROS depletion is used to evaluate a specific effect on a tissue or in biological systems. As a complement to these studies, our experimental approach implies a local and transient activation of in situ ROS production using the molecular machinery of the tissue. In this sense, our results are in close consonance with the observation that a transient ROS production occurs during tail regeneration in Xenopus tadpoles (Love et al., 2013). In this report, it is shown that activation of a regenerative signal, i.e., cutting the tadpole, is accompanied by a transient production of ROS. Here we demonstrate that switching on in situ transient ROS production activates a regenerative signal. It has also been reported that the mitochondrial transcription factor A (TFAM) is required for normal HF development and in vitro keratinocyte differentiation (Hamanaka et al., 2013), suggesting indirectly that mitochondrial ROS can have an important role in the regulation of skin function. Here we directly demonstrate that a transient generation of ROS in the mouse skin activates the HF stem cell population, promoting hair growth in the refractory telogen phase and accelerating burn healing. In this context, it would be interesting to evaluate the clinical potential of a transient ROS production in situ in the skin to stimulate tissue homeostasis—e.g., to improve the healing process of small burns and chronic wounds or to activate hair growth or prevent hair loss in certain types of alopecia.

We have found that switching on in situ ROS production by mALA-PT promotes a transient proliferative pulse in the tissue, including bulge cells, 2 days after the treatment. This proliferative burst results in extensive anagen development of HFs by days 8–10, showing extensive cell proliferation and Lef1 expression particularly in the hair germ, and subsequent hair growth by days 12–19 after mALA-PT. At day 2, most HFs are in telogen and, consequently, no evidence of β-catenin accumulation or transcriptional activation of Wnt/β-catenin signaling is observed. However, a significant activating phosphorylation of cSrc occurs at this time point. This result is particularly interesting, as no significant roles are usually assigned to Src kinase in relation to the regulation of skin stem cell function and/or the HF growth cycle. However, it has been reported that Src protein expression and activity are regulated during normal HF cycle (Serrels et al., 2009). In the same context, the molecular mechanisms regulating Src kinase activity by ROS in cultured cells are well characterized (Giannoni et al., 2010). These observations suggest that Src activity, independently or in combination with a controlled ROS production, can be an important partner in the skin signaling network, which merits further investigation. In this context, we hypothesize that mALA-PT induces, 2 days after treatment, an initial, cSrc-dependent, proliferative wave in the skin that is able to activate HF stem cell niches. This activation results in standard, Lef1-dependent, anagen entry around 6 days after treatments.

We have further shown that transiently switching on ROS production in the skin promotes a strong and unexpected transcriptional activation of prolactin family 2 subfamily c gene members, particularly Prl2c3. Surprisingly, we found that Prl2c3 was significantly translocated to the nucleus in most cell layers in the hyperplastic epithelium after mALA+Light treatment, suggesting that this hormone-like protein can have nuclear functions. Interestingly, it has been reported that this mitogen is expressed in the HF anagen growth phase (Fassett and Nilsen-Hamilton, 2001), and it facilitates the expansion of hematopoietic stem cells ex vivo (Choong et al., 2003). The role of Prlc3 in the skin is currently under investigation. In this context, the procedure presented here to transiently activate endogenous ROS production in the skin has been proven to be an efficient tool not only to suggest a physiological role for ROS in vivo but also to identify new signaling pathways and factors potentially implicated in the regulation of skin homeostasis.[/i]

That's not even a 5-head at this point, it's a 6-head.

idk, i saw many cute leonardos and giovannis when i went to Rome. tall, dark haired, dat "ciao bella" accent, hnng af.

Would be fcked if you wouldn't find any good looking guys in the capital, also many tourists there.

I mean what percent of Americans are actually fit? 5%? And this is probably <1% of Americans over 40. And then you add in other things that go against good looks (bad skin, bald etc.) and you get a small number of men who look good.

TBH though I think if a man keeps himself in top shape, he's going to at least look decent. Even if he loses hair, he can stay in shape and look pretty good. Worth noting that many old dudes also have 0/10 styling skills with hair. If you are going bald, either do something about it or embrace it. Walking around bald on top with hair on the sides looks dreadful.

Got any more of them Tinder at 40-50 stories to share?

where's the pic of the Reaper massacred this Chad

now

^ shiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiiitttttt tttttttt

dis real? friends and i had many lolz trolling on tinder with his pics. sad.

Lift and get a hair transplant. ZFG.

Also have a good personality, body language, sense of humor. Bonus points if you have a big dink.

Don't be a bitch.

full bald definitely looks fine if you're in shape. balding always looks awful unless you're 1 in a million like statham. there's a reason most of those celebrity bald shops are horseshoes and not full bald.you cant really compare a professionally done photo aided by stylists with him taking an off angle selfie looking like he's trying to solve a physics problem. focal length difference probably mattering most.

[QUOTE=gtg post_id=1628544073][img]https://3.bp.blogspot.com/-2zcit7S58T0/WiFlBE5d7zI/AAAAAAAABzE/C1s2bunTIkEzAK3XH1pPTNWq3DS20QnfgCPcBGAYYCw/s1600/IMG_0324.jpg[img]



now

[img]//assets.bodybuilding.com/forum/images/smilies/frown.gif[/img]
[img]https://i.redd.it/rexv009qqa861.png[img][/QUOTE]

did he decide to not sleep for 5 years or something wtf

Doing a quick search on him; he has 2 kids now.

And like someone else said, you're comparing an airbrushed photo with perfect lens to a selfie during day. This also doesn't include that he's not the same age in both photos.

Made my morning, Thanks!

Lol @ expecting to find a silver fox on Tinder. Fukn LOL!

I believe in taking care of myself and a balanced diet and rigorous exercise routine. In the morning if my face is a little puffy I’ll put on an ice pack while doing stomach crunches. I can do 1000 now. After I remove the ice pack I use a deep pore cleanser lotion. In the shower I use a water activated gel cleanser, then a honey almond body scrub, and on the face an exfoliating gel scrub. Then I apply an herb-mint facial mask which I leave on for 10 minutes while I prepare the rest of my routine. I always use an after shave lotion with little or no alcohol, because alcohol dries your face out and makes you look older. Then moisturizer, then an anti-aging eye balm followed by a final moisturizing protective lotion.”

Wisdom is strong in this one!



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